Tuesday, January 22, 2013

US home sales rose to 5-year high in 2012

(AP) ? U.S. sales of previously occupied homes rose in 2012 to their highest level in five years, spurred higher by record-low mortgage rates and steady hiring.

The National Association of Realtors said Tuesday that sales reached 4.65 million last year. That's up 9.2 percent from 2011 and the most since 2007. Sales are still below the roughly 5.5 million that are consistent with a healthy market.

Sales dipped in December to a pace of 4.94 million, down from 4.99 million in November. November's figure was revised lower, but was still the highest in three years.

The supply of available homes is shrinking as sales rise. That's pushing up prices. The inventory of homes for sale dropped to 1.82 million in December, the lowest in 12 years.

Still, the housing market is recovering and most analysts expect that should continue this year.

Steady hiring, record-low mortgage rates and a tight supply of new and previously occupied homes available for sale have helped boost sales and prices in most markets.

A measure of U.S. home prices rose 7.4 percent annually in November, real estate data provider CoreLogic said last week. That's the biggest annual increase since 2006, when the housing bubble began to burst. CoreLogic forecasts that home prices will rise 6 percent nationally this year.

Rising demand for homes has persuaded builders to step up construction, which adds to economic growth and hiring.

U.S. builders started work on homes in December at the fastest pace in 4 ? years, the government said last week. And builders finished 2012 as their best year for residential construction since the early stages of the housing crisis.

The gains in home building helped boost construction hiring in December by 30,000 jobs ? the most in 15 months.

Associated Press

Source: http://hosted2.ap.org/APDEFAULT/f70471f764144b2fab526d39972d37b3/Article_2013-01-22-US-Home-Sales/id-4b06d7bb90cd47db8761a558e68b683e

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Latinos take on more prominent role in Obama inauguration

WASHINGTON (AP) ? Latinos are taking a more prominent role in President Barack Obama's second inauguration, from the first Hispanic Supreme Court justice swearing in the vice president to a star-studded concert celebrating Latino culture.

Eva Longoria, a co-chairwoman for Obama's campaign, hosted "Latino Inaugural 2013: In Performance at the Kennedy Center" as a salute to the president Sunday evening ahead of his public swearing-in Monday. Jose Feliciano, Chita Rivera, Rita Moreno and Latin pop star Prince Royce all performed. The lineup also included Mario Lopez and Wilmer Valderrama.

Vice President Joe Biden and his family appeared onstage, drawing big cheers, to help open the show. He said he wanted to thank Latinos for their support in last year's election.

Biden said something profound happened with the enormous Latino support for Obama, and he said the Latino community underestimates its power.

"One thing that happened in this election, you spoke. You spoke in a way that the world, and I mean the world, as well as the United States, could not fail to hear," Biden said, calling the Latino vote decisive. "This is your moment. America owes you."

Feliciano opened the show by singing the national anthem.

Marc Anthony later drew big cheers when he applauded Latinos' growing political influence.

"Our united voice got us all here tonight and got the best man for the job in the White House," Anthony said.

San Antonio Mayor Julian Castro, who gave the keynote speech at last year's Democratic National Convention, recalled the admiration Latinos held for another president more than 50 years ago. Portraits of President John F. Kennedy still hang in many homes, he said.

"As we said 'Viva Kennedy' 50 years ago, today we say 'Viva Obama,'" Castro said.

A children's choir from San Juan, Puerto Rico, closed out the show, singing "This Land is Your Land." They were joined by a larger Latino choir, including Hispanic members of the U.S. military, in singing "America the Beautiful."

Earlier Sunday, Justice Sonia Sotomayor, an Obama appointee who is the first Hispanic justice on the highest court, administered the oath of office to Biden. And Richard Blanco, a son of Cuban exiles, is Obama's inauguration poet.

Latinos have a distinct presence at this inauguration after raising funds and turning out the vote for Obama in the 2012 election. Hispanics voted 7 to 1 for Obama over his challenger, Republican Mitt Romney, whose Hispanic support was less than any other presidential candidate in 16 years. Analysts said Romney's hardline stance on immigration was a factor.

San Antonio philanthropist and business leader Henry Munoz III, who coordinated the Latino inauguration event with Longoria and other Obama supporters, said this is a special moment when the Latino community is positioned to take an expanded role in shaping the country's future.

"Without question, the presidential election of 2012 proves that Latinos are perhaps the most important influence from this point forward in the election of the president of the United States," Munoz said. "It's important that the leadership in Washington view us not as a narrow interest group but as a vibrant political force" that carries not just votes, but influence and financial resources.

Organizers planned a series of symposiums, dinners and events ahead of the inauguration to keep people talking about issues that matter to Latinos, from immigration reform to building a Latino history museum on the National Mall. Munoz led a presidential commission that called on Congress in 2011 to authorize such a museum within the Smithsonian Institution, but Congress has not yet passed such a bill.

Munoz said it's important to keep Latinos engaged through the inauguration and beyond.

"Our work is not done. It doesn't end," he said. "We have a tendency to look at this phenomenon as ending on Election Day, when the reality is now it's time to get to work."

Longoria said this is her first inauguration. She has taken on a new role as political advocate since her days on "Desperate Housewives," pushing for a Latino history museum in Washington and raising funds for Obama's re-election.

Even though this is Obama's second inauguration, Longoria said there is still much to celebrate, including Sotomayor's role swearing in the vice president.

"There's something special about seeing a president recommit himself to the people of this great nation," she said before the show.

Longoria said she hopes to help influence policies, including immigration reform, and hopes Obama will make that his top priority as an economic issue. She called the Latino fundraising effort for the president a historic turning point.

"I think we have a permanent seat at the table, and now we're going to be able to have influence on what affects our communities," Longoria said. "I take civic responsibility very seriously, and I want to do what I can to help my country."

___

Follow Brett Zongker at https://twitter.com/DCArtBeat

Source: http://news.yahoo.com/latinos-bigger-role-obama-inauguration-202248106--politics.html

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Enzyme helps cancer cells avoid genetic instability

Enzyme helps cancer cells avoid genetic instability [ Back to EurekAlert! ] Public release date: 21-Jan-2013
[ | E-mail | Share Share ]

Contact: Rita Sullivan King
news@rupress.org
212-327-8603
Rockefeller University Press

Cancer cells are resourceful survivors with plenty of tricks for staying alive. Researchers have uncovered one of these stratagems, showing how cells lacking the tumor suppressor BRCA1 can resume one form of DNA repair, sparing themselves from stagnation or death. The study appears in the January 21st issue of The Journal of Cell Biology.

The BRCA1 protein helps to mend double-strand DNA breaks by promoting homologous recombination. Without it, cells can amass broken, jumbled, and fused chromosomes, which may cause them to stop growing or die. Although cells lacking BRCA1 seem like they should be vulnerable, loss of the protein instead seems to boost abnormal growth.

Recent studies have shown that cells lacking BRCA1 compensate by cutting back on 53BP1. This protein helps orchestrate a different DNA repair mechanism, nonhomologous end joining (NHEJ), and it thwarts a key step in homologous recombination. Researchers think that, in cells without BRCA1, 53BP1 spurs excessive NHEJ that can cause fatal chromosomal chaos. But with 53BP1 out of the way, the cells are able to resume homologous recombination. That might explain why cells that lack BRCA1 and eliminate 53BP1 can withstand traditional chemotherapy compounds and PARP inhibitors, a new generation of anti-cancer drugs that are in clinical trials. But how do cancer cells turn down 53BP1?

Researchers previously found that certain mutant fibroblasts increase production of cathepsin L, a protease that destroys 53BP1. BRCA1-deficient cancer cells take advantage of the same mechanism, according to a team of researchers led by Susana Gonzalo from the Washington University School of Medicine. When they cultured breast cancer cells that were missing BRCA1, the cells stopped growing. After two weeks of lethargy, however, some cells, which the researchers dubbed BOGA cells (BRCA1-deficient cells that overcome growth arrest), began to divide again. These cells showed increased levels of cathepsin L and reduced amounts of 53BP1. Eliminating cathepsin L from BOGA cells or dosing them with vitamin D, a cathepsin L inhibitor, prevented the decline in 53BP1 abundance.

To find out whether boosting cathepsin L levels enabled the cancer cells to restart homologous recombination, the researchers monitored sites of DNA damage tagged by RAD51, a protein that helps promote homologous recombination. The cells that had stopped growing did not display RAD51 foci, but these foci were prevalent in BOGA cells with reduced 53BP1. Removing cathepsin L from BOGA cells increased 53BP1 levels and diminished the number of RAD51 foci.

If cells can't perform homologous recombination, they turn to repair mechanisms such as NHEJ that can lead to jumbled chromosomes. However, after DNA-breaking doses of radiation, BOGA cells exhibited few chromosome defects. The number of these flaws climbed after the researchers stabilized 53BP1 levels by inhibiting cathepsin L or trimming its abundance.

The team then analyzed tumor samples from breast cancer patients. Researchers suspect that cathepsin L attacks 53BP1 by entering the nucleus. Samples from patients with BRCA1 mutations or with triple-negative breast canceran aggressive form of the diseaseshowed high levels of nuclear cathepsin L and reduced quantities of 53BP1. That suggests tumors in these patients hike the amounts of cathepsin L in the nucleus to break down 53BP1 and restore homologous recombination.

"It's a new pathway that explains how breast cancer cells lose 53BP1," says Gonzalo. How cancer cells boost nuclear cathepsin L levels is unclear, she notes.

Triple-negative breast cancers are currently identified by their lack of Her2 and the estrogen and progesterone receptors. The work suggests that another trio of measurementsthe amounts of 53BP1, cathepsin L, and vitamin D receptor in the nucleusmight help identify patients that are resistant to current breast cancer treatments. These people might respond to cathepsin inhibitors, some of which are undergoing animal testing. These compounds might steer the cells away from homologous recombination and leave them vulnerable to other therapies.

###

Grotsky, D.A., et al. 2013. J. Cell Biol. doi:10.1083/jcb.201204053.

About The Journal of Cell Biology (JCB) JCB is published by The Rockefeller University Press. All editorial decisions on manuscripts submitted are made by active scientists in conjunction with our in-house scientific editors. JCB content is posted to PubMed Central, where it is available to the public for free six months after publication. Authors retain copyright of their published works, and third parties may reuse the content for non-commercial purposes under a creative commons license. For more information, please visit www.jcb.org.


[ Back to EurekAlert! ] [ | E-mail | Share Share ]

?


AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.


Enzyme helps cancer cells avoid genetic instability [ Back to EurekAlert! ] Public release date: 21-Jan-2013
[ | E-mail | Share Share ]

Contact: Rita Sullivan King
news@rupress.org
212-327-8603
Rockefeller University Press

Cancer cells are resourceful survivors with plenty of tricks for staying alive. Researchers have uncovered one of these stratagems, showing how cells lacking the tumor suppressor BRCA1 can resume one form of DNA repair, sparing themselves from stagnation or death. The study appears in the January 21st issue of The Journal of Cell Biology.

The BRCA1 protein helps to mend double-strand DNA breaks by promoting homologous recombination. Without it, cells can amass broken, jumbled, and fused chromosomes, which may cause them to stop growing or die. Although cells lacking BRCA1 seem like they should be vulnerable, loss of the protein instead seems to boost abnormal growth.

Recent studies have shown that cells lacking BRCA1 compensate by cutting back on 53BP1. This protein helps orchestrate a different DNA repair mechanism, nonhomologous end joining (NHEJ), and it thwarts a key step in homologous recombination. Researchers think that, in cells without BRCA1, 53BP1 spurs excessive NHEJ that can cause fatal chromosomal chaos. But with 53BP1 out of the way, the cells are able to resume homologous recombination. That might explain why cells that lack BRCA1 and eliminate 53BP1 can withstand traditional chemotherapy compounds and PARP inhibitors, a new generation of anti-cancer drugs that are in clinical trials. But how do cancer cells turn down 53BP1?

Researchers previously found that certain mutant fibroblasts increase production of cathepsin L, a protease that destroys 53BP1. BRCA1-deficient cancer cells take advantage of the same mechanism, according to a team of researchers led by Susana Gonzalo from the Washington University School of Medicine. When they cultured breast cancer cells that were missing BRCA1, the cells stopped growing. After two weeks of lethargy, however, some cells, which the researchers dubbed BOGA cells (BRCA1-deficient cells that overcome growth arrest), began to divide again. These cells showed increased levels of cathepsin L and reduced amounts of 53BP1. Eliminating cathepsin L from BOGA cells or dosing them with vitamin D, a cathepsin L inhibitor, prevented the decline in 53BP1 abundance.

To find out whether boosting cathepsin L levels enabled the cancer cells to restart homologous recombination, the researchers monitored sites of DNA damage tagged by RAD51, a protein that helps promote homologous recombination. The cells that had stopped growing did not display RAD51 foci, but these foci were prevalent in BOGA cells with reduced 53BP1. Removing cathepsin L from BOGA cells increased 53BP1 levels and diminished the number of RAD51 foci.

If cells can't perform homologous recombination, they turn to repair mechanisms such as NHEJ that can lead to jumbled chromosomes. However, after DNA-breaking doses of radiation, BOGA cells exhibited few chromosome defects. The number of these flaws climbed after the researchers stabilized 53BP1 levels by inhibiting cathepsin L or trimming its abundance.

The team then analyzed tumor samples from breast cancer patients. Researchers suspect that cathepsin L attacks 53BP1 by entering the nucleus. Samples from patients with BRCA1 mutations or with triple-negative breast canceran aggressive form of the diseaseshowed high levels of nuclear cathepsin L and reduced quantities of 53BP1. That suggests tumors in these patients hike the amounts of cathepsin L in the nucleus to break down 53BP1 and restore homologous recombination.

"It's a new pathway that explains how breast cancer cells lose 53BP1," says Gonzalo. How cancer cells boost nuclear cathepsin L levels is unclear, she notes.

Triple-negative breast cancers are currently identified by their lack of Her2 and the estrogen and progesterone receptors. The work suggests that another trio of measurementsthe amounts of 53BP1, cathepsin L, and vitamin D receptor in the nucleusmight help identify patients that are resistant to current breast cancer treatments. These people might respond to cathepsin inhibitors, some of which are undergoing animal testing. These compounds might steer the cells away from homologous recombination and leave them vulnerable to other therapies.

###

Grotsky, D.A., et al. 2013. J. Cell Biol. doi:10.1083/jcb.201204053.

About The Journal of Cell Biology (JCB) JCB is published by The Rockefeller University Press. All editorial decisions on manuscripts submitted are made by active scientists in conjunction with our in-house scientific editors. JCB content is posted to PubMed Central, where it is available to the public for free six months after publication. Authors retain copyright of their published works, and third parties may reuse the content for non-commercial purposes under a creative commons license. For more information, please visit www.jcb.org.


[ Back to EurekAlert! ] [ | E-mail | Share Share ]

?


AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.


Source: http://www.eurekalert.org/pub_releases/2013-01/rup-ehc011813.php

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Obama girls to hit teen milestones in White House

President Barack Obama greets his daughter Sasha and Malia as first lady Michelle Obama watches at the ceremonial swearing-in at the U.S. Capitol during the 57th Presidential Inauguration in Washington, Monday, Jan. 21, 2013. (AP Photo/Evan Vucci)

President Barack Obama greets his daughter Sasha and Malia as first lady Michelle Obama watches at the ceremonial swearing-in at the U.S. Capitol during the 57th Presidential Inauguration in Washington, Monday, Jan. 21, 2013. (AP Photo/Evan Vucci)

President Barack Obama's daughters Malia Obama,left, and Sasha Obama arrive on the West Front of the Capitol in Washington, Monday, Jan. 21, 2013, for the Presidential Barack Obama's ceremonial swearing-in ceremony during the 57th Presidential Inauguration. (AP Photo/Win McNamee, Pool)

Sasha and Malia Obama arrive at the ceremonial swearing-in of their father President Barack Obama at the U.S. Capitol during the 57th Presidential Inauguration in Washington, Monday, Jan. 21, 2013. (AP Photo/Pablo Martinez Monsivais)

(AP) ? That's how it goes with kids. You hardly notice how fast they're growing up, then suddenly big sis is nearly as tall as Mom and the little one is a tween, gently sassing Dad.

On the inaugural platform again four years later, a more mature Malia Obama, 14, and Sasha, 11, smiled, sometimes giggled, and chatted with their cousin Avery Robinson as they awaited their father's arrival. Sasha bounced on her feet a bit as if chilly; later at the parade she danced in her seat to the beat of passing drummers. Malia, rivaling her mother's 5 feet 11 inches, looked poised in calf-high black boots. Like any girls their age, they whipped out their smartphones in the reviewing stand to take photos.

Both daughters appeared relaxed and oblivious to their global TV audience, unaffected by their rare status, unfazed by the fuss over their father.

Meanwhile, fashion-watchers were tweeting about the girls' coats in vibrant shades of purple. For the record: Malia wore a J. Crew ensemble, Sasha's was Kate Spade, and first lady Michelle Obama was in a Thom Browne coat with a navy print like a man's silk tie.

Such attention to the Obamas' clothes, their Hawaiian vacations, their hair ? Michelle lit up Twitter last week by adding bangs ? will continue as they charge into a time of turbulence for so many American families: the teen years.

In the second term Sasha, who arrived in the White House as a second-grader, moves on to high school. She expressed her pre-teen spirit Sunday, when Barack Obama took his official, nonpublic oath of office. After giving Dad a "Good job!" she added a reminder of his flubbed words four years ago. "You didn't mess up," Sasha teased the commander in chief.

For Malia, the milestones to come are many ? she'll be hitting the years when typical teens start driving, dating and applying to colleges. How normally can any of this go at 1600 Pennsylvania Ave.?

Life in the White House is bound to feel different to a teen than it does to a second-grader.

Seven-year-old Emanuel Coleman's grandmother positioned him on the steps of the National Gallery of Art to watch the swearing-in on a giant outdoor screen Monday. The Durham, N.C., boy thought life for a White House kid must be cool, because the president has "his own private limo, helicopter and lives in a really big house."

"It would be fun to fly in the presidential helicopter," Emanuel enthused.

Sixteen-year-old Colleen Casey isn't so sure.

"They have to live their life in their dad's shadow," said Casey, part of a group of Girl Scout volunteers who came to the inaugural from nearby Woodbridge, Va. "You can't be your own person."

That's the struggle for White House youngsters, said author Doug Wead, who has interviewed 19 sons and daughters of former presidents and wrote about them in "All the Presidents' Children."

"When your mom's the first lady, and all your classmates are oohing and ahhing over her, it's hard to compete with that," Wead said. "At any given time, half the country hates your father and half the country loves him. It's hard to establish a separate identity."

Just last week, the National Rifle Association referred to the Obama daughters in an ad berating their father for opposing a proposal to put armed guards in all schools, while his children get Secret Service protection. And the president's been criticized for sending Sasha and Malia to the private Sidwell Friends School.

Even the great stuff ? traveling the globe, meeting rock stars, mingling with world leaders ? can go to a girl's head.

Mrs. Obama says she strives to give the girls a normal life ? homecoming dances, playing basketball, trick-or-treating, slumber parties ? and also to keep them respectful, responsible and down-to-earth.

There's been lots of speculation that Mrs. Obama, who turns 50 next year, may design her own transformation in the second term, when she'll be freed from worries about her husband's re-election. Will the first lady who dubbed herself "mom-in-chief" add to her portfolio of family-centered causes? The White House isn't yet saying.

Some feminists want to see the Harvard Law School grad take on a more forceful public role. Not all her fans are so sure.

"I like the roles she's taken on with troops, with health, with children," said W. Faye Butts, 68, an enthusiastic Obama supporter who traveled from Macon, Ga., for the inaugural. No need to try to do more: "She has a family to raise, that's her first priority."

___

Associated Press writers Richard Lardner, Matthew Barakat and Darlene Superville contributed to this report.

___

Follow Connie Cass on Twitter: http://www.twitter.com/ConnieCass

Associated Press

Source: http://hosted2.ap.org/APDEFAULT/3d281c11a96b4ad082fe88aa0db04305/Article_2013-01-21-Inauguration-Family/id-eb303c7a22174d93a7c2da2d19face7e

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Loans Against Equipment In Construction Are Booming ? World ...

Loans Against Equipment In Construction Are Booming

?

VMI LLC, a nationwide asset-based funding source that provides flexible loan solutions for businesses with heavy equipment that is pledged as collateral, has just announced that it generated over $5 million in approvals during 2012. The final two quarters were especially strong for the company, which offers working capital loans as well as other loan services to its clients.

As the construction industry continues to gain strength, VMI LLC is anticipating an even larger year in 2013.

?We are incredibly optimistic about this upcoming year as traditional lenders aren?t lending but we are,? said Skylar Marshall, Communications Specialist from the equipment loan company, adding that since the loans are not credit-based, the staff at VMI LLC can work with clients whose credit scores are low.

?Traditional lenders have abandoned the commercial construction industry, but we are ready to provide construction companies with the working capital they need.?

As noted on the company?s website, one of the main reasons why businesses fail is due to undercapitalization. By working with VMI LLC and securing a loan against equipment, companies can get much-needed funding. Through the company?s construction equipment financing program, businesses can often secure equipment loans ranging from $10,000 up to $250,000. The company offers its equipment sale leaseback program to a variety of industries, including excavation, grading, demolition, paving, wrecking, hauling, and many more.

VMI LLC will typically finance heavy equipment of all varieties, including tractors, loaders, graders, excavators, cranes, and dump trucks. These often include well-known brands like Caterpillar, John Deere, Case, Komatsu, Hitachi, Kenworth and Peterbilt.

There are two main benefits to arranging for this type of business loan. First, by refinancing construction equipment, the business gets an inflow of cash that can be used for any business purpose they wish, including working capital, purchasing supplies, paying subcontractors or starting and completing projects.

Also, with the type of construction equipment refinance that is offered by VMI LLC, the equipment stays with the business so their projects can still be completed on time. There is no transfer of the equipment so there is no disturbance with the business, which is good news for both the company and its clients.

About VMI LLC:

VMI LLC provides secured construction equipment financing for companies that need working capital. Construction equipment funding is available for tractors, excavators, bulldozers, loaders, graders, scrapers, dump trucks, cranes, forklifts, backhoes, steers and other heavy equipment. Working capital loans are approved based on the equipment, not credit. For more information, please visit http://www.financeheavyequipment.com

Read the full story at SOURCE: http://www.prweb.com/releases/2013/1/prweb10341306.htm

Source: http://www.worldleasingnews.com/news/loans-against-equipment-in-construction-are-booming/

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Saturday, January 19, 2013

?The Yen Is Doomed?: For Japan, This Time Really ... - Yahoo! Finance

Despite all the talk about the U.S. fiscal cliff and debt ceiling, developments in Japan are shaping up to be 2013's biggest story for financial markets.

The yen has now fallen over 13% vs. the greenback since mid-November and traded below 90 to the dollar on Friday -- the first time since mid-2010. Shares of Japanese multinationals such as Toyota (TM) and Sony (SNE) have surged in reaction as a weaker yen makes them more competitive vs. their global competitors.

The yen?s weakness is a direct result of the campaign promises and policies of new Prime Minister Shinzo Abe, who won a landslide election in December.

Related: 'Obscene' Stimulus Will Trigger 'Made in Japan' Crisis in 2013: Mauldin

Thanks to a strong majority in Japan?s parliament and the pending retirement of three top Bank of Japan executives, Abe has a great opportunity -- and the political capital -- to live up to his promise to reinvigorate Japan?s economy by weakening the yen and embark on a massive stimulus program, according to Axel Merk, president of Merk Investments and manager of the Merk Funds, which has about $650 million of assets under management.

?The Japanese are known to talk, not act: That?s going to change,? Merk says.

Related: 2013 Could be the Year Japan?s Economy Turns Around: Cumberland?s Witherell

The yen has become known as the ?widow maker? for its historic propensity to punish those who?ve tried to short it. Similar to the dollar, the yen has enjoyed "safe haven" status during times of crisis. Repeatedly proven reports of its demise have been premature. But Merk believes this time really is different.

?The yen is doomed,? he says. ?We think the yen is going to drop substantially this year [and] eventually is going to be worthless.?

?Worthless? seems a bit hyperbolic and overly emotional. But Merk?s 2013 outlook piece cites the following fundamentals to support his forecast of continued weakness in the yen:

  • Abe?s government will appoint the three top positions at the Bank of Japan, as the governor and both deputy governors retire. Recent appointees have already been more dovish. Japanese culture is said to prefer talk over action, but the time for dovish talk may finally be over.
  • Despite its dovish reputation, the Bank of Japan barely expanded its balance sheet since 2008; of the major central banks only the Reserve Bank of Australia has been more hawkish.
  • Japan?s current account is sliding toward a deficit. That means, deficits will start to matter, eventually pushing up the cost of borrowing and making a 200%+ debt-to-GDP ratio unsustainable. (As you'll see in the accompanying video, Merk is quite animated on the subject of Japan's deteriorating current account deficit and views it as key to a bearish view on the yen.)
  • Abe?s government is as determined as it is blind. Abe believes a major spending program is just what Japan needs. As far as the yen is concerned, Abe may be getting far more than he is bargaining for.

Related: Japan?s Monetary Policies Are Disastrous for U.S. Economy: Peter Schiff

?The good news is we don?t think there?s a European crisis,? he says. ?There?s a shifting global crisis and it?s shifting from the U.S. to Europe to Japan.?

Regular viewers may see a pattern here. As you can see in the "related" links here, several guests have been spinning a variation of the same story in recent weeks: The yen is going to keep getting weaker and Abe?s government ? along with global markets ? should be careful what they wish for.

Aaron Task is the host of The Daily Ticker and Editor-in-Chief of Yahoo! Finance. You can follow him on Twitter at @aarontask or email him at altask@yahoo.com

Source: http://finance.yahoo.com/blogs/daily-ticker/yen-doomed-japan-time-really-different-merk-says-154750780.html

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How Berwyn Back Pain Relief Is Done Naturally Through ...

To successfully dispose of various types of pains that affect the body is the biggest concern of the Berwyn chiropractor. He is well skilled on ways to bring about relief to sufferers of pain using an unorthodox medical care called chiropractic. Backache is major among the numerous ailments which are brought to his office every day.

So many people suffer from back ache. As a matter of fact, it is common in all spheres of life. It is due to common factors such as stress, fatigue, poor posture and carrying very heavy objects. It begins as a slight pain, but if left unattended for long, it could result in excruciating pain levels that can even affect the ability to perform everyday normal activities.

As soon as aches are noticed in the back region, proper arrangement must be made to eradicate it. A chiropractic center could be visited in order to get the attention of the doctor. Other options may be sought also.

Back soreness starts off from the spinal cord which actually is the mid-point of every human musculoskeletal network. Pains result when some spine bones shift out of their standard position. This action causes tension to build up in the tendons, muscles, and nerves since the troubled bones exert a lot of pressure on the entire back region.

The factors earlier mentioned are largely responsible for shifting of bones in the spine. Out of them all, stress is the most critical. Quite a lot of persons carry on with work for hours unending without taking time out to rest in between. This is very harmful to general wellness because it leads to accumulation of stress. Body muscles begin to get stiffened and tired due to this and back pain is produced.

Chiropractic has several therapies for alleviating backache. These include laser therapy, spinal decompression, and sactive release. Another very effective therapy is massage. All these are natural techniques that completely take care of back pain. Besides these, health tips like resting properly, eating well, and exercising regularly are encouraged and taught by the Berwyn chiropractor.

Discover how chiropractic care helps relieve leg, back, hip and wrist pain safely, now. You can also find more information about a highly respected Berwyn chiropractor at http://www.creativechiropracticsolutions.com today.

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